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primary antibodies against βactin  (Santa Cruz Biotechnology)


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    Structured Review

    Santa Cruz Biotechnology primary antibodies against βactin
    Primary Antibodies Against βactin, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibodies+against+%CE%B2actin/anti+%CE%B2+actin/10__21203_slash_rs__3__rs___5364287_slash_v1-65-19-27
    Average 90 stars, based on 1 article reviews
    primary antibodies against βactin - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    other:

    Article Title: Vitamin D facilitates trophoblast invasion through induction of epithelial-mesenchymal transition.
    Article Snippet: Funding information A grant of the Korean Health Technology R&D Project, Ministry of Health & Welfare, Republic of Korea., Grant/Award Number: HI14C0368 Problem: Vitamin D deficiency increases the risk of developing pregnancyrelated complications, including preeclampsia and smallforgestationalage infants.. Vitamin D was demonstrated to promote the invasiveness of human extravillous trophoblasts (EVTs).. However, whether vitamin D induces the epithelial-mesenchymal transition (EMT) of EVTs remains unclear.

    Article Title: Pharmacological Activation of Peroxisome Proliferator-Activated Receptor {Delta} Increases Sphingomyelin Synthase Activity in THP-1 Macrophage-Derived Foam Cell.
    Article Snippet: Peroxisome proliferator-activated receptors (PPARs) are nuclear receptors, which mediate glucose and lipid homeostasis by regulating the expression of a large number of transcription factors.. Sphingomyelin synthase (SMS) is a key enzyme in the synthesis of sphingomyelin (SM), and its expression and activity have been reported to be associated with atherosclerosis (AS).. Although there have been many functional PPAR and SMS studies on atherosclerosis in recent years, few have investigated the correlation between the activation of PPARδ and the activity of SMS.

    Article Title: Functional analysis of GCNT3 for cell migration and EMT of castration-resistant prostate cancer cells.
    Article Snippet: Castration-resistant prostate cancer (CRPC) is a malignant tumor that is resistant to androgen deprivation therapy.. Treatments for CRPC are limited, and no diagnostic markers are currently available.. O-glycans are known to play an important role in cell proliferation, migration, invasion, and metastasis of cancer cells.

    Article Title: The Clustered, Regularly Interspaced, Short Palindromic Repeats-associated Endonuclease 9 (CRISPR/Cas9)-created MDM2 T309G Mutation Enhances Vitreous-induced Expression of MDM2 and Proliferation and Survival of Cells
    Article Snippet: The primary antibodies against βActin and the secondary antibodies of the horseradish peroxidase (HRP)-conjugated goat anti-rabbit IgG and anti-mouse IgG were purchased from Santa Cruz Biotechnology (Santa Cruz, CA).

    Incubation:

    Article Title: Attenuation of natural killer (NK) cell cytotoxicity by interaction between NKp30 of NK cells and dipeptidase 1 of colon cancer cells
    Article Snippet: .. Equal amounts of protein lysates were separated by SDS–PAGE, transferred to polyvinylidene di uoride membranes, and incubated with primary antibodies against βactin, glyceraldehyde-3-phosphate dehydrogenase (GAPDH), and His (Santa Cruz Biotechnology, Santa Cruz, CA, USA), DPEP1, PARP1, CD9, perforin, and granzyme B (Cell Signaling Technology, Danvers, MA, USA), and NKp30 (BioLegend, San Diego, CA, USA). .. Immunoreactive protein bands were visualized using an enhanced chemiluminescence detection reagent (EMD Millipore) and an Ez-Capture MG system (Atto Corporation, Japan).

    Western Blot:

    Article Title: Induction of chemokine CCL3 by NF-κB reduces methylmercury toxicity in C17.2 mouse neural stem cells.
    Article Snippet: Methylmercury is an environmental pollutant that shows selective toxicity to the central nervous system.. We previously reported that brain-specific expression of chemokine CCL3 increases in mice administered methylmercury.. However, the relationship between CCL3 and methylmercury toxicity has not been elucidated.



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